
Giuliana Clemente
I am currently a postdoctoral researcher at the University of Manchester in the lab of Dr Thomas Millard. My research goal is to define mechanism(s) of cell migration and routing in complex in vivo settings using the humble yet powerful fruit fly.
I completed my pre-doctoral studies at the University of Dundee (Scotland) under the supervision of Professor Arno Müller. During my PhD, being fascinated by cytoskeleton dynamics during cell division and cell migration, I contributed to the characterisation of the mitotic and post-mitotic functions of Drosophila Spindly.
Mitophagy protects beta cells from inflammatory damage in diabetes
Selected by | Giuliana Clemente |
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Cell clusters adopt a collective amoeboid mode of migration in confined non-adhesive environments.
Selected by | Giuliana Clemente |
Polarized endosome dynamics engage cytosolic Par-3 and dynein during asymmetric division
Selected by | Giuliana Clemente |
Macrophages transfer mitochondria to sensory neurons to resolve inflammatory pain
Selected by | Giuliana Clemente |
Modulation of neuronal resilience during aging by Hsp70/Hsp90/STI1 chaperone system
Selected by | Giuliana Clemente |
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A conserved MFS orchestrates a subset of O-glycosylation to facilitate macrophage dissemination and tissue invasion
Selected by | Giuliana Clemente |
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PIKfyve/Fab1 is required for efficient V-ATPase and hydrolase delivery to phagosomes, phagosomal killing, and restriction of Legionella infection
Selected by | Giuliana Clemente |
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Aurora A depletion reveals centrosome-independent polarization mechanism in C. elegans
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Centrosome Aurora A gradient ensures a single PAR-2 polarity axis by regulating RhoGEF ECT-2 localization in C. elegans embryos
Selected by | Giuliana Clemente |
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A novel mechanism of gland formation in zebrafish involving transdifferentiation of renal epithelial cells and live cell extrusion
Selected by | Giuliana Clemente |
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Human macrophages survive and adopt activated genotypes in living zebrafish
Selected by | Giuliana Clemente |
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Spatiotemporally controlled Myosin relocalization and internal pressure cause biased cortical extension to generate sibling cell size asymmetry
Selected by | Giuliana Clemente |